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The mechanism involves PRPs growth factorsparticularly PDGF, VEGF, and IGF-1stimulating the proliferation of dermal papilla cells, improving scalp vascularity, prolonging the anagen (growth) phase of the hair cycle, and reducing the inflammation that drives follicular miniaturization in androgenetic alopecia

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HGF and c-met inhibitors in anticancer therapy The c-Met is not only a marker linked to metastatic properties but is also a suitable drug target and the molecules which inhibit HGF/c-Met signaling are expected to serve for therapeutic intervention [51]